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Price: $100.00
10,000 mcg/Vial
Cyanocobalamin B12 is a synthetic form of Vitamin B12, essential for maintaining healthy nerve cells and red blood cells. Unlike methylcobalamin, cyanocobalamin needs to be converted by the body into its active forms before it can be used. Despite this, it is highly effective in correcting B12 deficiencies and supporting overall health. Cyanocobalamin B12 is commonly used to boost energy levels, support neurological function, and enhance mood and cognitive abilities.
Boosts Energy Levels – Aids in red blood cell production to help increase energy and combat fatigue.
Supports Neurological Health – Maintains the protective covering of nerves, contributing to overall neurological health.
Enhances Mood and Cognitive Function – Participates in neurotransmitter synthesis to help improve mood and cognitive performance.
Administered via injection to ensure optimal absorption and effectiveness. Follow your healthcare provider’s guidance for dosing frequency.
Vitamin B12 injections bypass the digestive system, providing direct absorption into the bloodstream. This method is particularly beneficial for individuals with absorption issues, ensuring that the body receives the full benefits of this vital nutrient.
Once injected, cyanocobalamin is absorbed into the bloodstream and converted to its active forms, such as methylcobalamin and adenosylcobalamin. This conversion allows it to participate in critical bodily functions like energy production, red blood cell formation, and DNA synthesis.
Cyanocobalamin supports the conversion of food into energy by aiding in the metabolism of fats and proteins. This process helps produce ATP (adenosine triphosphate), ensuring a steady energy supply for daily activities and physical exertion.
Cyanocobalamin contributes to the maintenance and regeneration of nerve cells, supporting cognitive function and mood stability. By facilitating the synthesis of neurotransmitters, it helps prevent neurological disorders and promotes mental clarity.
This form of B12 helps reduce homocysteine levels, lowering the risk of heart disease. It supports the production of red blood cells and improves blood flow, contributing to overall cardiovascular health.
"The customer support team was incredibly helpful and knowledgeable about B12 injections. They made sure I understood everything and were always available for follow-up questions. I've felt a noticeable boost in energy and focus."
Sophia G.
"Since starting cyanocobalamin injections, I've experienced faster recovery times and less muscle soreness after workouts. The service was great, and the quality of the product exceeded my expectations."
Jake M.
"My strength and endurance have improved significantly with cyanocobalamin B12 injections. I can train harder and recover faster. The detailed consultation and high-quality service were outstanding."
Lucas F.
"Dealing with fatigue and brain fog was tough, but cyanocobalamin injections have helped clear my mind and boost my focus. The service was excellent, and I felt well cared for throughout."
Maya L.
"As an avid runner, I noticed improved stamina and quicker recovery times after starting B12 injections. The personalized consultation ensured I received the right dosage for my needs."
Carlos R.
"Cyanocobalamin B12 injections have made a significant difference in my overall well-being. I feel more energetic and vibrant. The service was efficient and professional."
Ava S.
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In most cases, methylcobalamin is nontoxic, even in large doses. Adverse reactions reported following methylcobalamin administration include headache, infection, nausea/vomiting, paresthesias, and rhinitis. Adverse reactions following intramuscular (IM) injection have included anxiety, mild transient diarrhea, ataxia, nervousness, pruritus, transitory exanthema, and a feeling of swelling of the entire body. Some patients have also experienced a hypersensitivity reaction following intramuscular injection that has resulted in anaphylactic shock and death. In cases of suspected cobalt hypersensitivity, an intradermal test dose should be administered.
During the initial treatment period with methylcobalamin, pulmonary edema and congestive heart failure have reportedly occurred early in treatment with parenteral methylcobalamin. This is believed to result from the increased blood volume induced by methylcobalamin. Peripheral vascular thrombosis has also occurred. In post-marketing experience, angioedema and angioedema-like reactions were reported with parenteral methylcobalamin.
Hypokalemia and thrombocytosis could occur upon conversion of severe megaloblastic anemia to normal erythropoiesis with methylcobalamin therapy. Therefore, monitoring of the platelet count and serum potassium concentrations are recommended during therapy. Polycythemia vera has also been reported with parenteral methylcobalamin.
Diarrhea and headache.
Call your health care provider immediately if you are experiencing any signs of an allergic reaction: skin rash, itching or hives, swelling of the face, lips, or tongue, blue tint to skin, chest tightness, pain, difficulty breathing, wheezing, dizziness, red, swollen painful area on the leg.
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Our dedicated network of board-certified clinicians ensures lab test reviews and consultations are completed in a compliant, efficient, and customer-friendly manner. We ensure your customers receive clinically appropriate test reviews and consultations. Through our user-friendly patient-doctor platform, practitioners can easily submit requisitions electronically, improving efficiency. This seamless process enhances practitioner collaboration, leading to faster and more accurate test results.
Our Methylcobalamin B12 Injections are cash pay as we do not offer support for medical insurance at this time.
Who should not take this medication? Patients with early hereditary optic nerve atrophy, cyanocobalmin hypersensitivity, and those who are pregnant. Your health care provider needs to know if you have any of these conditions: kidney disease; Leber’s disease; megaloblastic anemia; an unusual or allergic reaction to methylcobalamin, cobalt, other medicines, foods, dyes, or preservatives; pregnant or trying to get pregnant; breastfeeding.
Methylcobalamin is contraindicated in patients with methylcobalamin hypersensitivity or hypersensitivity to any of the medication components. Methylcobalamin is also contraindicated in patients with cobalt hypersensitivity because methylcobalamin contains cobalt. In the case of suspected cobalt hypersensitivity, an intradermal test dose should be administered because anaphylactic shock and death have followed parenteral administration of methylcobalamin.
Methylcobalamin should not be used in patients with early hereditary optic nerve atrophy (Leber’s disease). Optic nerve atrophy can worsen in patients whose methylcobalamin levels are already elevated. Hydroxocobalamin is the preferred agent in this patient population (see separate monograph in Less Common Drugs).
Most formulations of methylcobalamin injection contain benzyl alcohol as a preservative. Benzyl alcohol may cause allergic reactions. Methylcobalamin injections should be used cautiously in those patients with benzyl alcohol hypersensitivity. Methylcobalamin, vitamin B12 preparations containing benzyl alcohol should be avoided in premature neonates because benzyl alcohol has been associated with ‘gasping syndrome,’ a potentially fatal condition characterized by metabolic acidosis and CNS, respiratory, circulatory, and renal dysfunction.
Vitamin B12 deficiency can suppress the symptoms of polycythemia vera. Treatment with methylcobalamin or hydroxocobalamin may unmask this condition.
Folic Acid, vitamin B9 is not a substitute for methylcobalamin, vitamin B12 deficiency, although it may improve vitamin B12 megaloblastic anemia. However, exclusive use of folic acid in treating vitamin B12 deficient megaloblastic anemia could result in progressive and irreversible neurologic damage. Before receiving folic acid or methylcobalamin, patients should be assessed for deficiency and appropriate therapy started concurrently. The intranasal formulations are not approved to treat acute B12 deficiency; all hematologic parameters should be normal before beginning the methylcobalamin intranasal formulations. Concurrent iron-deficiency anemia and folic acid deficiency may result in a blunted or impeded response to methylcobalamin therapy.
Certain conditions may blunt or impede therapeutic response to methylcobalamin therapy. These include serious infection, uremia or renal failure, drugs with bone marrow suppression properties (e.g., chloramphenicol), or concurrent undiagnosed folic acid or iron deficiency anemia. The mechanism appears to be interference with erythropoiesis. Patients with vitamin B12 deficiency and concurrent renal or hepatic disease may require increased doses or more frequent administration of methylcobalamin.
Clinical reports have not identified differences in responses between elderly and younger patients. Generally, dose selection for elderly patients should be done with caution. Elderly patients tend to have a greater frequency of decreased hepatic, renal, or cardiac function, and also have concomitant disease or receiving other drug therapy. Start with doses at the lower end of the dosing range.
Vitamin B12, or methylcobalamin, is essential to growth, cell reproduction, hematopoiesis, and nucleoprotein and myelin synthesis. Cells characterized by rapid division (epithelial cells, bone marrow, myeloid cells) appear to have the greatest requirement for methylcobalamin. Vitamin B12 can be converted to coenzyme B12 in tissues; in this form it is essential for conversion of methylmalonate to succinate and synthesis of methionine from homocysteine (a reaction which also requires folate). In the absence of coenzyme B12, tetrahydrofolate cannot be regenerated from its inactive storage form, 5-methyl tetrahydrofolate, resulting in functional folate deficiency. Vitamin B12 also may be involved in maintaining sulfhydryl (SH) groups in the reduced form required by many SH-activated enzyme systems. Through these reactions, vitamin B12 is associated with fat and carbohydrate metabolism and protein synthesis. Vitamin B12 deficiency results in megaloblastic anemia, GI lesions, and neurologic damage (which begins with an inability to produce myelin and is followed by gradual degeneration of the axon and nerve head). Vitamin B12 requires an intrinsic factor-mediated active transport for absorption, therefore, lack of or inhibition of intrinsic factor results in pernicious anemia.
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YOURx cannot guarantee refunds for any cancellation requests received after the pharmacy has processed your order. Furthermore, YOURx does not accept returns nor guarantee refunds for any product once it has left the pharmacy.
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